Susanne Schreiber

The paper analyzes mitochondrial morphology and spatial organization in Drosophila neurons to identify systematic rules governing their positioning and cell-type specificity.

The paper shows that the electrogenicity of the Na+/K+-ATPase can cause firing shifts and disrupt computation in highly active spiking cells.

The paper shows that calcium channel isoform diversity increases variance in gating properties, enhancing neuronal excitability robustness to perturbations.

The paper develops a reduced neuron model that captures bifurcation structure rather than voltage traces, preserving physiological parameter dependence in class I conductance-based models.

The paper shows that weak synaptic coupling and intrinsic cellular dynamics can generate rich network states with enhanced variability and information encoding in balanced networks.

The paper shows that dendritic morphology affects neuronal spiking dynamics and network synchronisation states through mathematical modeling.

The paper demonstrates that slow neuronal rhythms can arise from the interplay of voltage dynamics and extracellular potassium concentration changes, mediated by the Na+/K+-ATPase, without requiring synaptic connectivity or special ion channels.

The paper identifies feed-forward inhibition as a key mechanism regulating vocal response timing in zebra finches using a spiking network model and electrophysiological recordings.

This paper introduces a dynamic clamp protocol to artificially modify cell capacitance in neurons, demonstrating its feasibility in both models and in vitro experiments.

The paper identifies a neural circuit mechanism in insect flight where electrical synapses de-synchronize motoneurons to produce stable wingbeat power.

The paper proposes a computational model of systems memory consolidation based on Hebbian plasticity in parallel synaptic pathways, showing how memories are transferred and transformed across brain regions.

The paper shows that intense neuronal activity changes ionic concentrations, leading to a switch in firing patterns and excitability types in mouse cortical neurons.