Christian J. Janzen

This study identifies novel Leishmania virulence factors through proteomics of infected macrophages and presents a database for further research.

This study shows that DOT1B is not essential for differentiation or macrophage infection in Leishmania mexicana, contrasting its essential role in Trypanosoma brucei.

The paper identifies TelAP2 as an interacting partner of TelAP1 in the telomere complex of Trypanosoma brucei and shows that TelAP1 requires this interaction to associate with telomeric proteins.