Author

P. Hande Özdi̇nler

Northwestern UniversityORCID 0000-0003-4125-6013Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2

Frequent Co-Authors

Abraham Acevedo‐ArozenaiD· 1 shared Aksu GünayiD· 1 shared Angela Ahrens· 1 shared Aurea B. Martins-BachiD· 1 shared Barış GençiD· 1 shared Benjamin HelmoldiD· 1 shared Brian J. NiemaniD· 1 shared Dennis Keefe· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperNeuroscience

Abraham Acevedo‐ArozenaORCID

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2 papers

Recent·Most cited

NeuroscienceNeurobiology of DiseasePeer-reviewedFeb 15, 2024

TDP-43-M323K causes abnormal brain development and progressive cognitive and motor deficits associated with mislocalised and increased levels of TDP-43.

Juan Miguel Godoy-Corchuelo, Zeinab Ali, José Miguel Brito Armas…·Silvia Corrochano

This study characterizes a mouse model with TDP-43 M323K mutation, showing cognitive, motor, and structural brain abnormalities linked to TDP-43 mislocalization and increased levels.

10 citations

mouseexperimental assay

𝕏 PostBluesky

NeurosciencebioRxivPreprintOct 4, 2022

SBT-272 improves TDP-43 pathology in the ALS motor cortex by modulating mitochondrial integrity, motility, and function

Mukesh Gautam, Barış Genç, Benjamin Helmold…·P. Hande Özdi̇nler

This study shows that SBT-272 improves mitochondrial structure, motility, and function in TDP-43 pathology of ALS motor neurons, reducing inflammation and preserving neuron health.

DOI 10.1101/2022.10.04.510854

mouseexperimental assay

𝕏 PostBluesky