Author

John R. Corboy

University of Colorado AnschutzORCID 0000-0003-4169-1461Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Andrew F. TeichiD· 1 shared Annie LeeiD· 1 shared Anthony KhairallahiD· 1 shared Bradley T. HymaniD· 1 shared Bridget Shafit‐ZagardoiD· 1 shared Charles C. WhiteiD· 1 shared David Alan BennettiD· 1 shared Gilad Green· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperNeuroscience

Andrew F. TeichORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintJun 4, 2022

A cross-disease human microglial framework identifies disease-enriched subsets and tool compounds for microglial polarization.

John F. Tuddenham, Mariko F. Taga, Verena Claudia Haage…·Philip L. De Jager

This study identifies disease-enriched microglial subsets and validates compounds that modulate their states using single-cell RNA sequencing and in vitro experiments.

20 citations

humanscRNA-seq

𝕏 PostBluesky

NeurosciencebioRxivPreprintMay 5, 2022

TMEM106B is increased in Multiple Sclerosis plaques, and deletion causes accumulation of lipid after demyelination

Bridget Shafit‐Zagardo, Simone Sidoli, James E. Goldman…·Rashed M. Nagra

This study identifies TMEM106B as an insoluble protein increased in MS plaques and shows its deletion causes axonal damage and lipid accumulation in demyelination models.

2 citations

mouseproteomics

𝕏 PostBluesky