Author

Jyothi Arikkath

University of Nebraska Medical CenterORCID 0000-0003-4246-2767Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Neuroscience· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:proteomics· 1

Frequent Co-Authors

Rahmi LeeiD· 2 shared Seonil KimiD· 2 shared Ava A. Steger· 1 shared Emma S. Hinchliffe· 1 shared Hadassah Mendez-VazqueziD· 1 shared Jessica P. RobertsiD· 1 shared Kaila Nip· 1 shared Madeleine C. MoseleyiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersNeuroscience

Rahmi LeeORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

NeuroscienceNeurosciencePeer-reviewedFeb 5, 2026

δ-catenin haploinsufficiency is sufficient to alter behaviors and glutamatergic synapses in mice

Emma S. Hinchliffe, Victoria Aragon, Van T. Mai…·Seonil Kim

This study shows that δ-catenin haploinsufficiency in mice disrupts social behavior and fear learning, and alters glutamatergic synaptic activity.

DOI 10.1016/j.neuroscience.2026.01.044

mouseexperimental assay

𝕏 PostBluesky

NeuroscienceProceedings of the National Academy of SciencesPeer-reviewedMay 22, 2023

The autism-associated loss of δ-catenin functions disrupts social behaviors

Hadassah Mendez-Vazquez, Regan L. Roach, Kaila Nip…·Seonil Kim

This paper investigates how the G34S mutation in δ-catenin causes loss of function leading to social dysfunction in ASD by altering glutamatergic activity and GSK3β-mediated degradation.

15 citations

mouseproteomics

𝕏 PostBluesky