Author

Sabin Dhakal

Montreal Clinical Research InstituteORCID 0000-0003-4257-7986Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Developmental Biology· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Approach:statistical model· 3 Research Focus:differential expression· 2 Organism:mouse· 2 Assay:scRNA-seq· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1

Frequent Co-Authors

Gilberto Duran-Bishop· 2 shared Loïck JoumieriD· 2 shared Mohan K. MalleshaiahiD· 2 shared Muhammad UsamaiD· 2 shared Sanidhya Jagdish· 2 shared Élie LambertiD· 1 shared François RobertiD· 1 shared Rajni JaiswaliD· 1 shared

Activity by Year

2017-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersCell Biology

Gilberto Duran-Bishop

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3 papers

Recent·Most cited

Cell BiologyFrontiers in Cell and Developmental BiologyPeer-reviewedJan 23, 2026

BMP and NODAL Paracrine Signalling Regulate the Totipotent-like Cell State in Embryonic Stem Cells.

Sanidhya Jagdish, Loïck Joumier, Sabin Dhakal…·Mohan K. Malleshaiah

This study identifies BMP and NODAL signalling as critical for regulating totipotent-like cell states in embryonic stem cells through cell-cell communication.

1 citation

mousescRNA-seq

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Developmental BiologybioRxivPreprintOct 14, 2025

Induction of cellular totipotency by NACC1-driven feed-forward regulation.

Thulaj Meharwade, Gilberto Duran-Bishop, Loïck Joumier…·Mohan K. Malleshaiah

The paper identifies NACC1 as a regulator of totipotent-like cells in mouse embryonic stem cells through a feed-forward mechanism involving gene regulation and retrotransposon activity.

DOI 10.1101/2025.10.14.682353

mousescRNA-seq

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Systems BiologybioRxivPreprintAug 20, 2017

Inferring propensity amongst lung and breast carcinomas via overlapped gene expression profiles

Rajni Jaiswal, Sabin Dhakal, Shaurya Jauhari

The paper identifies overlapping gene expression profiles between lung and breast carcinomas to find common hub genes that could serve as potential drug targets.

DOI 10.1101/178558

humanbulk RNA-seq

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