Author

Oyedele Adewale Adeyi

University of Alabama at BirminghamORCID 0000-0003-4272-1095Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell-cell communication· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Organism:mouse· 2 Assay:spatial transcriptomics· 2 Approach:statistical model· 2 Assay:ATAC-seq· 1 Organism:C. elegans· 1

Frequent Co-Authors

Jeffrey H. AlbrechtiD· 2 shared Laura J. NiedernhoferiD· 2 shared Linshan LauxiD· 2 shared Maggie Klaers· 2 shared Aaron P. HavasiD· 1 shared Adam Burgess· 1 shared Adam LewisiD· 1 shared Akhil AmbekariD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCell Biology

Jeffrey H. AlbrechtORCID

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3 papers

Recent·Most cited

Cell BiologybioRxivPreprintAug 10, 2026

Cellular senescence is associated with age-related loss of liver zonation and hepatocyte function

Linshan Laux, Alisha Aristel, Syed Ali…·Laura J. Niedernhofer

This study shows that cellular senescence disrupts liver zonation and hepatocyte function with age by altering gene expression and cell signaling.

DOI 10.64898/2026.08.08.743614

mousespatial transcriptomics

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PhysiologybioRxivPreprintMay 28, 2026

Cyclin D1 regulates the hepatic response to feeding: Evidence for non-cell cycle roles in the liver

Heng Wu, Jonathan I Hauser, Na Yang…·Jeffrey H. Albrecht

This study shows cyclin D1 regulates hepatic gene networks and metabolic responses to feeding, with non-cell cycle roles in aging, MASLD, and C. elegans overnutrition.

1 citation

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Cell BiologyKidney InternationalPeer-reviewedApr 27, 2026

A MEF2C transcription factor network regulates proliferation of glomerular endothelial cells in diabetic kidney disease

Ricardo Melo Ferreira, Debora L. Gisch, Carrie L. Phillips…·Tashas Cameron-Wheeler

This study identifies a MEF2C transcription factor network that regulates endothelial cell proliferation in diabetic kidney disease and shows that SGLT2i treatment can reverse its dysregulation.

6 citationstop 1%

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