Scott E. Evans

The paper shows that inhibiting apoptosis of alveolar myofibroblasts (AMFs) reprograms them into ductal myofibroblasts (DMFs) in a mouse model, suggesting fate plasticity in lung mesenchymal cells.

The paper identifies AP-1 transcription factors as regulators of a transitional state in lung alveolar type 2 cells during injury-repair, linking chromatin changes to both regeneration and tumorigenesis.

This study shows that nebulized Toll-like receptor agonists improve survival and immune function in a mouse model of influenza-associated pulmonary aspergillosis.

The paper introduces OBIF, a systems model that identifies molecular drivers of synergy by integrating statistical and biological interactions from omics data.