David J. Schulz

The paper presents a genome and transcriptome annotation of Cancer borealis, revealing neuronal gene structure and transcript diversity with high confidence.

This paper shows that stomach muscles of Cancer borealis hyperpolarize with warming and operate optimally within a narrow pH window, mediated by K2P channels that reduce excitability and maintain neuromuscular function under environmental change.

This study shows that EAE alters bladder function and MPG neuron properties, with more severe disease leading to broader action potentials and reduced ion channel expression.

The paper develops a biophysical model of a crustacean cardiac ganglion cell to explore morphology and conductance constraints using a rejection protocol and experimental data.

The paper examines the synaptic contributions of pacemaker neurons in the Jonah crab's pyloric network, finding that the glutamatergic AB neuron contributes more than the cholinergic PD neuron.

The paper evaluates how accurately transcriptional profiling can classify neuronal cell types using small crustacean ganglia, finding that expression profiles alone are insufficient without additional modalities.

This study examines how type II diabetes affects neuronal excitability and ion channel expression in female mice's major pelvic ganglion neurons over time.

The paper investigates how dopamine maintains network synchrony in crab cardiac ganglion neurons by modulating gap junctions, preventing desynchronization caused by serotonin or K+ channel blockers.

The paper examines how chronic spinal cord injury alters gene co-expression networks of ion channels and receptors in mouse lumbar spinal cord, revealing cell-type-specific changes in correlation patterns.