Author

Annamaria Carissimo

Istituto per le Applicazioni del Calcolo Mauro PiconeORCID 0000-0003-4636-3969Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Genetics· 1 Synthetic Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

Nicola Brunetti‐PierriiD· 2 shared Alessandro BelvedereiD· 1 shared Alessandro PalmaiD· 1 shared Alessia GrandiosoiD· 1 shared Antonio RinaldiiD· 1 shared Christian MuelleriD· 1 shared Daniela TrisciuoglioiD· 1 shared Diego VozziiD· 1 shared

Activity by Year

2020-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersSynthetic Biology

Nicola Brunetti‐PierriORCID

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3 papers

Recent·Most cited

Cancer BiologyCell Death and DiseasePeer-reviewedNov 6, 2025

A circuit involving the lncRNA MB3 and the driver genes MYC and OTX2 inhibits apoptosis in Group 3 Medulloblastoma by regulating the TGF-β pathway via HMGN5

Alessia Grandioso, Paolo Tollis, Francesca Romana Pellegrini…·Pietro Laneve

2 citations

humanexperimental assay

𝕏 PostBluesky

Synthetic BiologyNature CommunicationsPeer-reviewedAug 4, 2025

Computational identification of small molecules for increased gene expression by synthetic circuits in mammalian cells

M. Pisani, F Calandra, Antonio Rinaldi…·Velia Siciliano

The paper identifies small molecules that enhance gene expression in synthetic circuits by analyzing RNA-seq data and drug-induced profiles.

10 citations

humanbulk RNA-seq

𝕏 PostBluesky

GeneticsbioRxivPreprintMay 4, 2020

CHOP-c-JUN complex plays a critical role in liver proteotoxicity induced by mutant Z alpha-1 antitrypsin.

Sergio Attanasio, Gwladys Gernoux, Rosa Ferriero…·Nicola Brunetti‐Pierri

The paper identifies the CHOP-c-JUN complex as a key driver of liver proteotoxicity in AAT deficiency by enhancing SERPINA1 transcription and ATZ accumulation.

1 citation

𝕏 PostBluesky