Author

Renyta Moses

University of PennsylvaniaORCID 0000-0003-4708-2958Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Gregory Kelly· 2 shared Kara N. MaxwelliD· 2 shared Ryan M. HausleriD· 2 shared A Dinh Le· 1 shared Abigail G. DoucetteiD· 1 shared Alexandra IndegliaiD· 1 shared Alison Schwartz LevineiD· 1 shared Anne NaumeriD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersGenetics

Gregory Kelly

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2 papers

Recent·Most cited

GeneticsNature CommunicationsPeer-reviewedMay 21, 2026

Distinct genomic and immunologic tumor evolution in germline TP53-driven breast cancers

Nabamita Boruah, David Hoyos, Renyta Moses…·Kara N. Maxwell

This study compares genomic and immunologic features of germline TP53-driven breast cancers to sporadic breast cancers, finding distinct patterns of genomic instability and immune infiltration.

4 citations

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologyMolecular Cancer ResearchPeer-reviewedMay 18, 2026

Variation at the R181 residue of p53 confers loss of p53 DNA binding cooperativity with the retention of mitochondrial-associated apoptosis

Renyta Moses, Alexandra Indeglia, Alison Schwartz Levine…·Kara N. Maxwell

This study shows that p53 variants with R181 mutations retain mitochondrial apoptosis function but lose DNA binding cooperativity, leading to reduced cancer risk.

1 citation

humanDNA methylation

𝕏 PostBluesky