Author

Gregory Kelly

University of Pennsylvania

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Kara N. MaxwelliD· 2 shared Renyta MosesiD· 2 shared Ryan M. HausleriD· 2 shared A Dinh Le· 1 shared Abigail G. DoucetteiD· 1 shared Alexandra IndegliaiD· 1 shared Alison Schwartz LevineiD· 1 shared Anne NaumeriD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersGenetics

Kara N. MaxwellORCID

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2 papers

Recent·Most cited

Distinct genomic and immunologic tumor evolution in germline TP53-driven breast cancers

Nabamita Boruah, David Hoyos, Renyta Moses, Ryan M. Hausler, Heena Desai, A Dinh Le, Madeline Good, Gregory Kelly, Ashvathi Raghavakaimal, Maliha N Tayeb, Mohana Narasimhamurthy, Abigail G. Doucette, Peter Edward Gabriel, Michael D. Feldman, Jinae Park, , Kara N. Maxwell

This study compares genomic and immunologic features of germline TP53-driven breast cancers to sporadic breast cancers, finding distinct patterns of genomic instability and immune infiltration.

Nature Communications · May 21, 2026Peer-reviewedGenetics

Variation at the R181 residue of p53 confers loss of p53 DNA binding cooperativity with the retention of mitochondrial-associated apoptosis

Renyta Moses, Alexandra Indeglia, Alison Schwartz Levine, Ryan M. Hausler, Gregory Kelly, Sven A. Miller, Isabel Anez-Bruzual, Melissa Heller, R Delgado, Caitlin Orr, Wendy Kohlmann, Anne Naumer, Jennie Vagher, Sophie R Cahill, Luke Devon Maese, , Kara N. Maxwell

This study shows that p53 variants with R181 mutations retain mitochondrial apoptosis function but lose DNA binding cooperativity, leading to reduced cancer risk.

Molecular Cancer Research · May 18, 2026Peer-reviewedCancer Biology