Diego Clemente

The paper shows that disease severity in a murine MS model correlates with oligodendrocyte production and that M-MDSCs are associated with increased oligodendrocyte generation.

This study identifies TAF1-dependent transcriptional dysregulation as a mechanism underlying multiple sclerosis progression and presents an in vivo model for therapeutic development.

The paper identifies myeloid-derived suppressor cells (M-MDSCs) as biomarkers for predicting milder clinical courses in multiple sclerosis.

This study shows that TLR4-pathway activation in astrocytes after TBI leads to synaptic loss and BBB damage, which can be mitigated by TLR4 inhibition.