Alberto Pérez‐Samartín

This study identifies TAF1-dependent transcriptional dysregulation as a mechanism underlying multiple sclerosis progression and presents an in vivo model for therapeutic development.

This study investigates the migratory behavior of enucleated and non-enucleated cells, showing that locomotion is regulated by systemic, self-organized dynamics.

The paper quantitatively analyzes cell migration trajectories across species and stimuli, finding that directional movement results from systemic, integrative cellular processes rather than individual molecular components.

The paper shows that sub-clinical doses of NMDA and AMPA glutamate receptor antagonists protect against demyelination in experimental models of multiple sclerosis.

This study shows clemastine increases oligodendrocyte differentiation but impairs myelination during development, likely due to microglia-oligodendrocyte interactions.

This study demonstrates associative memory in Amoeba proteus by linking a conditioned stimulus (electric field) with a non-conditioned stimulus (chemotactic peptide), showing learned systemic motility patterns.

This study investigates whether the nucleus controls migration in Amoeba proteus by comparing enucleated and non-enucleated cells, finding no direct nuclear control over movement patterns.