Author

Shreyasi Dey Sarkar

Saha Institute of Nuclear PhysicsORCID 0009-0000-0383-1115Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biophysics· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:chromatin & epigenetic regulation· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Kaushik SenguptaiD· 2 shared Amrutayani Panda· 1 shared Ayan BanerjeeiD· 1 shared Bhabani Shankar MohantyiD· 1 shared Duhita SenguptaiD· 1 shared Kakoli BoseiD· 1 shared Mithun Kumar MitraiD· 1 shared Poulami Goswami· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCancer Biology

Kaushik SenguptaORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintSep 10, 2026

Remodelling of chromatin architecture and super-enhancer landscape in lamin A/C depleted HGSOC

Shreyasi Dey Sarkar, Poulami Goswami, Amrutayani Panda…·Kaushik Sengupta

This study investigates the effects of lamin A/C depletion on chromatin organization in HGSOC, finding detethering of gene-poor chromatin, compartment switching, and rewiring of super-enhancers, leading to reduced proliferation.

DOI 10.64898/2026.09.07.749802

humanbulk RNA-seq

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BiophysicsNucleic Acids ResearchPeer-reviewedJun 13, 2025

Cardiomyopathic mutations of lamin A perturb mutual interactions of lamin, nuclear membrane, and chromatin leading to LLPS

Subhradip Nath, Shuvadip Dutta, Shreyasi Dey Sarkar…·Kaushik Sengupta

This study shows that cardiomyopathic lamin A mutations disrupt nuclear structure and chromatin organisation, leading to liquid-like aggregates and genome spatial disorganisation.

9 citations

humanimaging

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