Author

Subhradip Nath

Saha Institute of Nuclear PhysicsORCID 0000-0003-2703-2822Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biophysics· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:experimental assay· 1 Assay:imaging· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

Kaushik SenguptaiD· 2 shared Debasish Prusty· 1 shared Duhita SenguptaiD· 1 shared Mithun Kumar MitraiD· 1 shared Shreyasi Dey SarkariD· 1 shared Shuvadip DuttaiD· 1 shared Sk Ramiz IslamiD· 1 shared Soumen K. MannaiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

8 top collaborators · 16 mutual co-author links

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2 shared papersCell Biology

Kaushik SenguptaORCID

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintJul 17, 2025

Defects in lamin A-Prohibitin crosstalk leads to ROS elevation and OxPhos imbalance in laminocardiomyopathy

Subhradip Nath, Soumen K. Manna, Debasish Prusty…·Kaushik Sengupta

This study shows that the lamin A K97E mutation disrupts PHB2 interaction, causing mitochondrial dysfunction, ROS elevation, and metabolic imbalance, contributing to dilated cardiomyopathy.

DOI 10.1101/2025.07.17.665267

humanmetabolomics

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BiophysicsNucleic Acids ResearchPeer-reviewedJun 13, 2025

Cardiomyopathic mutations of lamin A perturb mutual interactions of lamin, nuclear membrane, and chromatin leading to LLPS

Subhradip Nath, Shuvadip Dutta, Shreyasi Dey Sarkar…·Kaushik Sengupta

This study shows that cardiomyopathic lamin A mutations disrupt nuclear structure and chromatin organisation, leading to liquid-like aggregates and genome spatial disorganisation.

9 citations

humanimaging

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