Author

Fengting Su

Universität InnsbruckORCID 0009-0001-6430-9221Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:metabolomics· 2 Organism:C. elegans· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

Andreas KoeberleiD· 2 shared Amelie LuppiD· 1 shared André GollowitzeriD· 1 shared Armando ZarrelliiD· 1 shared Ehsan Bonyadi Rad· 1 shared Felix J. Benscheid· 1 shared Giovanni Di FabioiD· 1 shared Handan Melike DönertaşiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

Andreas KoeberleORCID

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2 papers

Recent·Most cited

Cell BiologyNature CommunicationsPeer-reviewedApr 18, 2026

Aging-associated decline of phosphatidylcholine synthesis is a malleable trigger of natural mitochondrial aging.

Tetiana Poliezhaieva, Yuting Li, Prerana Shrikant Chaudhari…·Maria Ermolaeva

The paper identifies a decline in phosphatidylcholine synthesis as a driver of mitochondrial aging that can be reversed by dietary interventions in both worms and human cells.

10 citationstop 2%

C. eleganshuman

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Pharmacology & ToxicologyTheranosticsPeer-reviewedJan 6, 2025

Silybin A from Silybum marianum reprograms lipid metabolism to induce a cell fate-dependent class switch from triglycerides to phospholipids

Solveigh C. Koeberle, Maria Thürmer, Markus Werner…·Andreas Koeberle

This study identifies silybin A's role in reprogramming lipid metabolism to shift from triglycerides to phospholipids in liver cells, with implications for liver protection.

11 citations

humanmouse

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