Author

Michael P. Schonfeld

University of Kansas Medical CenterORCID 0009-0001-7393-6876Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Organism:C. elegans· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

Benjamin R. ThomsoniD· 1 shared Edward Benjamin ThorpiD· 1 shared Gabriella S. Borkowski· 1 shared Ganeshkumar Rajendran· 1 shared Guillaume CourboniD· 1 shared Heng Wu· 1 shared Irina TikhanovichiD· 1 shared James O’Sullivan· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperMolecular Biology

Benjamin R. ThomsonORCID

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2 papers

Recent·Most cited

PhysiologybioRxivPreprintMay 28, 2026

Cyclin D1 regulates the hepatic response to feeding: Evidence for non-cell cycle roles in the liver

Heng Wu, Jonathan I Hauser, Na Yang…·Jeffrey H. Albrecht

This study shows cyclin D1 regulates hepatic gene networks and metabolic responses to feeding, with non-cell cycle roles in aging, MASLD, and C. elegans overnutrition.

1 citation

mousehuman

𝕏 PostBluesky

Molecular BiologyJournal of Clinical InvestigationPeer-reviewedDec 2, 2024

Post-ischemic inactivation of HIF prolyl hydroxylases in endothelium promotes maladaptive kidney repair by inducing glycolysis

Ratnakar Tiwari, Rajni Sharma, Ganeshkumar Rajendran…·Pinelopi P. Kapitsinou

This study shows that inactivating PHD enzymes in endothelial cells after ischemic kidney injury promotes maladaptive repair through glycolysis and MCT4, which can be reversed by MCT4 inhibitors.

28 citationstop 2%

mousehuman

𝕏 PostBluesky