Author

James O’Sullivan

Northwestern University

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Biology Disciplines

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Molecular Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Benjamin R. ThomsoniD· 1 shared Edward Benjamin ThorpiD· 1 shared Gabriella S. Borkowski· 1 shared Ganeshkumar Rajendran· 1 shared Guillaume CourboniD· 1 shared Matthew John SchipmaiD· 1 shared Michael P. SchonfeldiD· 1 shared Navdeep S. ChandeliD· 1 shared

Activity by Year

2024-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperMolecular Biology

Benjamin R. ThomsonORCID

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1 paper

Recent·Most cited

Post-ischemic inactivation of HIF prolyl hydroxylases in endothelium promotes maladaptive kidney repair by inducing glycolysis

Ratnakar Tiwari, Rajni Sharma, Ganeshkumar Rajendran, Gabriella S. Borkowski, Si Young An, Michael P. Schonfeld, James O’Sullivan, Matthew John Schipma, Yalu Zhou, Guillaume Courbon, Benjamin R. Thomson, Valentin David, Susan E. Quaggin, Edward Benjamin Thorp, Navdeep S. Chandel, Pinelopi P. Kapitsinou

This study shows that inactivating PHD enzymes in endothelial cells after ischemic kidney injury promotes maladaptive repair through glycolysis and MCT4, which can be reversed by MCT4 inhibitors.

Journal of Clinical Investigation · Dec 2, 2024Peer-reviewedMolecular Biology