Author

Sarah M Butts

University of VirginiaORCID 0009-0002-0106-2812Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:benchmarking· 1 Research Focus:cancer & tumor microenvironment· 1 Assay:clinical cohort / trial· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

Andrew W. Schaefer· 2 shared Elias T. SpiliotisiD· 2 shared Franklin Mayca PozoiD· 2 shared Cristina MontagnaiD· 1 shared Ilona A. Kesisova· 1 shared Md Noor A. AlamiD· 1 shared Priyanka BhaktiD· 1 shared Sarath Reghunathan· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

9 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

Andrew W. Schaefer

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Septin crosstalk with microtubules and actin is regulated by a GSK3-dependent phosphoswitch

Md Noor A. Alam, TrishaJean C Holt, Andrew W. Schaefer, Franklin Mayca Pozo, Sarath Reghunathan, Sarah M Butts, Priyanka Bhakt, Ilona A. Kesisova, Elias T. Spiliotis

This paper identifies GSK3-dependent phosphorylation of SEPT9 as a mechanism regulating its partitioning between actin and microtubules, affecting neuronal polarization.

bioRxiv · May 9, 2026 · PreprintCell Biology

Septins buffer actomyosin forces to protect the nucleus from genotoxic mechanical stress

Franklin Mayca Pozo, Sarah M Butts, Andrew W. Schaefer, Cristina Montagna, Elias T. Spiliotis

This study shows septins buffer actomyosin forces to protect the nucleus from mechanical stress during migration and in non-migrating cells, with SEPT9 amplification linked to reduced genomic damage and higher mortality in breast cancer.

bioRxiv · Jan 21, 2026 · PreprintCell Biology