Author

Luke Dorrian

Boston UniversityORCID 0009-0002-1640-183XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Benjamin L. WoloziniD· 2 shared Alejandro Nico Rondón-OrtiziD· 1 shared Alexander KnyshoviD· 1 shared Alper Kilci· 1 shared Chelsea Joy WebberiD· 1 shared Guangxin SuniD· 1 shared Jenna LiberaiD· 1 shared Jingjing SuniD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersNeuroscience

Benjamin L. WolozinORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintMay 2, 2025

Loss of age-associated increase in m6A-modified RNA contributes to GABAergic dysregulation in Alzheimer's disease

Jenna Libera, Junming Hu, Tuyet-anh Nguyen…·Benjamin L. Wolozin

The paper shows that loss of age-related m6A RNA methylation in Alzheimer's disease brains is linked to GABAergic dysfunction and synaptic issues.

5 citations

humanbulk RNA-seq

NeurosciencebioRxivPreprintMar 28, 2023

Accumulation of m6A exhibits stronger correlation with MAPT than beta-amyloid pathology in an APP NL-G-F /MAPT P301S mouse model of Alzheimer's disease

Lulu Jiang, Rebecca Roberts, Melissa Wong…·Benjamin L. Wolozin

This study shows that m6A accumulation correlates more strongly with MAPT pathology than beta-amyloid in an Alzheimer's mouse model.

5 citations

mouseexperimental assay