Author

Gema María Esteban-Ortega

Consejo Superior de Investigaciones CientíficasORCID 0009-0002-2010-1549Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:mouse· 1 Research Focus:protein structure prediction· 1 Assay:proteomics· 1

Frequent Co-Authors

Margarita Díaz‐GuerraiD· 2 shared Elena Torres-CamposiD· 1 shared Gonzalo S. TejedaiD· 1 shared Lola Ugalde‐TriviñoiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

4 top collaborators · 4 mutual co-author links

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2 shared papersNeuroscience

Margarita Díaz‐GuerraORCID

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2 papers

Recent·Most cited

NeuroscienceCell Death and DiseasePeer-reviewedAug 29, 2025

Retrograde transport of neurotrophin receptor TrkB-FL induced by excitotoxicity regulates Golgi stability and is a target for stroke neuroprotection

Gema María Esteban-Ortega, Elena Torres-Campos, Margarita Díaz‐Guerra

The paper demonstrates that excitotoxicity-induced TrkB-FL downregulation is due to retrograde transport to the Golgi and shows that a neuroprotective peptide can interfere with this process to preserve Golgi stability and neuronal survival in stroke.

9 citations

experimental assay

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NeuroscienceTheranosticsPeer-reviewedMar 21, 2025

Interactions established by isoform-specific TrkB-T1 sequences govern inflammatory response and neurotoxicity in stroke

Lola Ugalde‐Triviño, Gonzalo S. Tejeda, Gema María Esteban-Ortega, Margarita Díaz‐Guerra

The paper identifies TrkB-T1 isoform-specific protein interactions and shows that interfering with them reduces neuroinflammation and neurotoxicity in a mouse stroke model.

6 citations

mouseproteomics

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