Author

Masayuki Umeda

St. Jude Children's Research HospitalORCID 0009-0002-5636-6602Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Research Focus:perturbation response prediction· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Jeffery M. KlcoiD· 2 shared Stanley B PoundsiD· 2 shared Alex KentsisiD· 1 shared Ali KhalighifariD· 1 shared Anna L. W. HuskeyiD· 1 shared Aurelie ClaquiniD· 1 shared Bappaditya ChandraiD· 1 shared Benjamin A. GarciaiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersGenetic & Genomic Medicine

Jeffery M. KlcoORCID

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2 papers

Recent·Most cited

Genetic & Genomic MedicinemedRxivPreprintNov 4, 2025

Clonal Evolution of Pediatric Acute Myeloid Leukemia and Its Contribution to Disease Relapse

Masayuki Umeda, Pandurang S. Kolekar, Anna L. W. Huskey…·Jeffery M. Klco

This study analyzes genetic changes in pediatric AML relapse using whole-genome sequencing and finds that relapse is driven by the selection of pre-existing clones rather than new mutations.

1 citation

humanwhole-genome sequencing

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Cancer BiologyCancer DiscoveryPeer-reviewedJun 19, 2025

MOZ and HBO1 Histone Acetyltransferase Complexes Are Molecular Dependencies and Therapeutic Targets in NUP98-Rearranged Acute Myeloid Leukemia

Nicole Lynn Michmerhuizen, Emily Heikamp, Ilaria Iacobucci…·Charles Grenfell Mullighan

This study identifies MOZ and HBO1 as molecular dependencies in NUP98-rearranged AML, showing their inhibition reduces leukemic burden and synergizes with Menin inhibitors.

29 citationstop 1%

mouseproteomics

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