Author

Shuai Hong

University Medical Center Hamburg-EppendorfORCID 0009-0003-6159-5386Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Birgit Schwanke· 2 shared Durga Praveen MekaiD· 2 shared Froylán Calderón de AndaiD· 2 shared Karun Kar SinghiD· 2 shared Nadeem MurtazaiD· 2 shared Annie A. Cheng· 1 shared Bhooma ThiruvahindrapuramiD· 1 shared Bradley W. DobleiD· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersNeuroscience

Birgit Schwanke

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Direct control of translational elongation by TAOK2β highlights altered protein synthesis as a fundamental underlying component of autism

Melad Henis, Tabitha Rücker, Robin Scharrenberg, Melanie Richter, Lucas L. Baltussen, Shuai Hong, Durga Praveen Meka, Birgit Schwanke, Nagammal Neelagandan, Danie Daaboul, Nadeem Murtaza, Christoph Krisp, Sönke Harder, Hartmut Schlüter, Matthias Kneussel, , Froylán Calderón de Anda

The paper identifies TAOK2β as a regulator of translational elongation and shows its disruption leads to increased protein synthesis in an ASD mouse model.

Science Advances · Apr 12, 2024Peer-reviewedNeuroscience

Neuron-specific protein network mapping of autism risk genes identifies shared biological mechanisms and disease relevant pathologies

Nadeem Murtaza, Annie A. Cheng, Chad O. Brown, Durga Praveen Meka, Shuai Hong, Jarryll A. Uy, Joelle El-Hajjar, Neta Pipko, Brianna K. Unda, Birgit Schwanke, Sansi Xing, Bhooma Thiruvahindrapuram, Worrawat Engchuan, Brett Trost, Éric Deneault, , Karun Kar Singh

The paper maps neuron-specific protein networks of autism risk genes, identifies shared biological mechanisms, and links them to disease-relevant pathologies.

Cell Reports · Nov 1, 2022Peer-reviewedNeuroscience