Froylán Calderón de Anda

This study maps the molecular changes in murine upper-layer cortical neurons during development and shows how maternal immune activation disrupts this process through post-transcriptional and signaling mechanisms.

The paper identifies molecular pathways associated with neurodevelopmental disorder subgroups using neuroimaging data from mice and humans.

The paper identifies TAOK2β as a regulator of translational elongation and shows its disruption leads to increased protein synthesis in an ASD mouse model.

The paper maps neuron-specific protein networks of autism risk genes, identifies shared biological mechanisms, and links them to disease-relevant pathologies.

This study shows that Scrib deletion in mice causes cortical thinning, disrupted connectivity, and psychomotor deficits, linking Scrib to neurodevelopmental disorders.

The paper shows radial F-actin organization around the centrosome during early neuronal development, with dynamic actin structures and interactions with centrosomal components.

The paper shows that Tao kinase regulates synaptic growth and connectivity in Drosophila nociceptive circuits to maintain specificity and function during larval development.