Laure Fronzaroli‐Molinières

This study shows that brief monocular deprivation induces homeostatic changes in intrinsic neuronal excitability in thalamic relay cells, involving Kv1 and Nav1 channel regulation and electrical coupling.

This study shows that visual activity increases intrinsic excitability in thalamic relay neurons through LTP-IE, involving calcium influx and PKA activation, and links this to visual plasticity and amblyopia.

This study shows that expressing LGI1 in LGI1-deficient neurons restores normal excitability by restoring Kv1.1 channel density and function at the axon initial segment.

The paper shows that up-regulation of synaptic strength and intrinsic excitability in CA3 neurons is linked to regulation of axonal Kv1.1 channels following synaptic activity deprivation.

The paper shows axonal sodium channels transmit input synchrony levels to postsynaptic cells by modulating action potential amplitude, enhancing synaptic transmission for synchronous inputs.