Jorge Ramírez‐Franco

The paper identifies a hyperglycosylated and phosphorylated form of Kv1.2 upregulated in Lgi1-/- mice and reports changes in its interactome.

This study shows that enhancing astrocytic Kir4.1 function reduces spasticity in SCI mice by improving K+ uptake and preventing neuronal hyperexcitability.

This study identifies α2δ1 as an extracellular GluA1 interactor that regulates LTP, spatial memory, and seizure susceptibility through its role in AMPAR trafficking.

The paper identifies and characterizes a conserved population of spinal cerebrospinal fluid contacting neurons in mice, revealing their sensory functions and receptor expression.

The paper uses deep learning to identify a DAT-negative subpopulation of dopaminergic neurons in the lateral substantia nigra in mouse and human brain sections.

This study shows that expressing LGI1 in LGI1-deficient neurons restores normal excitability by restoring Kv1.1 channel density and function at the axon initial segment.

This study investigates Kv1 channel downregulation in LGI1 knock-out mice, showing it occurs in somatodendritic regions and is linked to increased neuronal bursting and changes in the Kv1 interactome.

The paper uses patient-derived autoantibodies to study LGI1 localization and interactome in mouse brains, revealing its synaptic distribution and novel protein complexes.

This study investigates the role of Kv4 channels in SNc DA neurons, showing Kv4.2 also contributes to IA and demonstrating IA gating kinetics and IH interactions determine pacemaking and post-inhibitory responses.