Marco Taddei-Tardon

This study investigates the molecular mechanisms of serotonergic psychedelics using a neural stem cell model, revealing an integrated 5-HT2A-TrkB signaling network involved in neuroplasticity and metabolic responses.

This study uses quantitative phosphoproteomics to show that hallucinogenic psychedelics induce distinct global phosphorylation patterns compared to non-hallucinogenic analogues, with functional relevance to glycolytic metabolism.