Jessica L. Wager

The paper identifies hyper-proliferation of adipose progenitors in offspring of obese mothers as a mechanism for early-onset obesity and increased adipocyte number.

This study identifies redox signaling and IL-6 as key regulators of adipogenesis in adipose progenitor cells under obesogenic conditions.

This study shows that estrogen protects female mice from adipose progenitor cell exhaustion during diet-induced obesity, but this protection is lost after ovariectomy.

This study shows that high maternal adiposity during pregnancy increases offspring susceptibility to diet-induced hepatosteatosis, with lipidomic changes linked to intrauterine environment effects.

This study shows that reactive oxygen species (ROS) mediate the pro-adipogenic effects of bisphenol A and its analogues by enhancing adipogenesis through mitochondrial ROS production.