J. von Kempis

The paper identifies a ciliogenesis-primed secretory cell state preferentially infected by seasonal H1N1 IAV in human airway epithelium.

This study shows avian IAVs preferentially bind ciliated cells but replicate similarly in secretory and ciliated cells, while human IAVs bind ciliated cells but replicate more efficiently in secretory cells due to ANP32 usage.

The paper compares nasal and bronchial mucus from human airway cultures to identify components that influence influenza A virus inhibition.