Author

Jeremy Lins

Brown UniversityORCID 0009-0007-0528-5307Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Organism:C. elegans· 2 Approach:experimental assay· 2

Frequent Co-Authors

Anne C. HartiD· 2 shared Adam FriedbergiD· 1 shared Burak UnsaliD· 1 shared Diana A. WalliD· 1 shared Jonah SimoniD· 1 shared Kelsey N. SchuchiD· 1 shared Natalie ChapkisiD· 1 shared Roza KhalifaiD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

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10 top collaborators · 25 mutual co-author links

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2 shared papersNeuroscience

Anne C. HartORCID

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2 papers

Recent·Most cited

NeuroscienceDisease Models & MechanismsPeer-reviewedMay 1, 2026

C. elegans models of Alternating Hemiplegia of Childhood have dominant neuromuscular junction defects

Diana A. Wall, Adam Friedberg, Jeremy Lins…·Anne C. Hart

The paper introduces C. elegans models of Alternating Hemiplegia of Childhood (AHC) by introducing AHC-causing ATP1A3 mutations into the orthologous gene, revealing dominant neuromuscular junction defects.

1 citation

C. elegansexperimental assay

𝕏 PostBluesky

NeurosciencebioRxivPreprintOct 11, 2019

Disrupted Autophagy and Neuronal Dysfunction in C. elegans Knockin Models of FUS Amyotrophic Lateral Sclerosis

Saba Baskoylu, Natalie Chapkis, Burak Unsal…·Anne C. Hart

This study uses C. elegans knock-in models to show that autophagy dysfunction contributes to neuronal defects in FUS-related ALS, independent of SQSTM1/p62 function.

2 citations

C. elegansexperimental assay

𝕏 PostBluesky