Author

Jing Li

Rush University Medical CenterORCID 0009-0007-4228-0003Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Jing Li25 papers · Chinese Academy of Sciences

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:scRNA-seq· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

Aadil AhmediD· 1 shared Adrian Pablo MansiniiD· 1 shared Bethany E. Perez WhiteiD· 1 shared Christian Fernando Guerrero‐JuareziD· 1 shared Christopher D. NazaroffiD· 1 shared Christopher McCraeiD· 1 shared Connor ColeiD· 1 shared Enno SchmidtiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

Aadil AhmedORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

ImmunologyNature CommunicationsPeer-reviewedAug 6, 2025

IgG autoantibodies in bullous pemphigoid directly induce a pathogenic MyD88-dependent pro-inflammatory response in keratinocytes:

Lei Bao, Christian Fernando Guerrero‐Juarez, Jing Li…·Kyle T. Amber

This study shows that BP-IgG autoantibodies induce a pro-inflammatory response in keratinocytes via MyD88-dependent signaling, contributing to BP pathology.

11 citationstop 3%

humanbulk RNA-seq

𝕏 PostBluesky