Author

Enno Schmidt

Philipps University of MarburgORCID 0000-0002-1206-8913Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 2 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 3 Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

Aadil AhmediD· 1 shared Adrian Pablo MansiniiD· 1 shared Aleksa ČepićiD· 1 shared Alexandra Holscher· 1 shared Aude Zimmerman· 1 shared Bethany E. Perez WhiteiD· 1 shared Christian Fernando Guerrero‐JuareziD· 1 shared Christoph HolscheriD· 1 shared

Activity by Year

2025-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

Aadil AhmedORCID

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

γδ T cell-derived IL-17A aggravates experimental epidermolysis bullosa acquisita in response to IL-1β

Fengyuan Deng, Sandra Jukic, Melanie Wannick, Alexandra Holscher, Immo Prinz, Ralf J Ludwig, Enno Schmidt, Xinhua Yu, Frank Petersen, Christoph Holscher

This study shows that γδ T cell-derived IL-17A worsens experimental EBA, and IL-1β drives IL-17A production in these cells.

bioRxiv · Nov 6, 2026 · PreprintImmunology

Gut Microbiota Modulates and Predicts Disease Severity in Experimental Pemphigoid Disease

Xiaolin Liu, Sabrina Patzelt, Yue Ma, Aleksa Čepić, Hasan Onur Dikmen, Katja Bieber, Mathieu Groussin, Mathilde Poyet, Saleh Mohamed Ibrahim, Enno Schmidt, John F. Baines

This study shows gut microbiota influences disease severity in an experimental pemphigoid model, with specific taxa linked to protection or inflammation.

bioRxiv · Oct 1, 2025 · PreprintMicrobiology

IgG autoantibodies in bullous pemphigoid directly induce a pathogenic MyD88-dependent pro-inflammatory response in keratinocytes:

Lei Bao, Christian Fernando Guerrero‐Juarez, Jing Li, Manuela Pigors, Shirin Emtenani, Yingzi Liu, Adrian Pablo Mansini, Yulu F. Wang, Aadil Ahmed, Norito Ishii, Takashi Hashimoto, Bethany E. Perez White, Stefan J. Green, Kevin Kunstman, Nicole C. Nowak, , Kyle T. Amber

This study shows that BP-IgG autoantibodies induce a pro-inflammatory response in keratinocytes via MyD88-dependent signaling, contributing to BP pathology.

Nature Communications · Aug 6, 2025Peer-reviewedImmunology

SERPINB5-TGF-β signalling modulates desmoplakin membrane localization and ameliorates pemphigus vulgaris skin blistering

Maitreyi Rathod, Mariam Petrosyan, Aude Zimmerman, Maike Märker, Tobias Gosau, Henriette Franz, Tomás Cunha, Enno Schmidt, Michael Hertl, Volker Spindler

This study shows that SERPINB5 modulates TGF-β signaling to stabilize desmoplakin and prevent pemphigus vulgaris blistering.

Annals of Anatomy - Anatomischer Anzeiger · Jun 1, 2025Peer-reviewedCell Biology