Author

Olivia K. Heintz

Boston Children's HospitalORCID 0009-0007-6946-0450Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:proteomics· 1

Frequent Co-Authors

Fernanda Soares Rasquel-OliveiraiD· 2 shared Michael S. RogersiD· 2 shared Victor FattoriiD· 2 shared Waldiceu Aparecido Verri JriD· 2 shared Anne E. Lindholm· 1 shared Ashish Kumar JainiD· 1 shared Cynthia KohliD· 1 shared Daniëlle PeterseiD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersNeuroscience

Fernanda Soares Rasquel-OliveiraORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintOct 20, 2025

Pro-endometriosis macrophage release of IL-33 is key for endometriosis pain and lesion formation

Victor Fattori, Fernanda Soares Rasquel-Oliveira, Soledad Ochoa…·Michael S. Rogers

This study identifies IL-33 as a key mediator in endometriosis pain and lesion formation through neuroimmune communication involving CGRP and macrophages.

2 citations

mousehuman

𝕏 PostBluesky

NeurosciencebioRxivPreprintAug 28, 2023

Nociceptor to macrophage communication through CGRP/RAMP1 signaling drives endometriosis-associated pain and lesion growth

Victor Fattori, Tiago H. Zaninelli, Fernanda Soares Rasquel-Oliveira…·Michael S. Rogers

This study identifies CGRP/RAMP1 signaling as a driver of endometriosis pain and lesion growth, showing that blocking this pathway reduces pain and lesion size in mice.

2 citations

mousehuman

𝕏 PostBluesky