Author

Tiago H. Zaninelli

Universidade Estadual de LondrinaORCID 0000-0001-7233-477XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

Michael S. RogersiD· 2 shared Olivia K. HeintziD· 2 shared Raymond M. AnchaniD· 2 shared Victor FattoriiD· 2 shared Waldiceu Aparecido Verri JriD· 2 shared Allison F. VitonisiD· 1 shared Anne E. Lindholm· 1 shared Ashish Kumar JainiD· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersPharmacology & Toxicology

Michael S. RogersORCID

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2 papers

Recent·Most cited

Pharmacology & ToxicologyJournal of Advanced ResearchPeer-reviewedAug 12, 2024

Targeting NGF but not VEGF or BDNF signaling reduces endometriosis-associated pain in mice

Tiago H. Zaninelli, Victor Fattori, Olivia K. Heintz…·Michael S. Rogers

This study shows that blocking NGF-TrkA signaling reduces endometriosis-associated pain in mice, while blocking VEGF-VEGFR1 or BDNF-TrkB does not.

24 citationstop 2%

mouseproteomics

𝕏 PostBluesky

NeurosciencebioRxivPreprintAug 28, 2023

Nociceptor to macrophage communication through CGRP/RAMP1 signaling drives endometriosis-associated pain and lesion growth

Victor Fattori, Tiago H. Zaninelli, Fernanda Soares Rasquel-Oliveira…·Michael S. Rogers

This study identifies CGRP/RAMP1 signaling as a driver of endometriosis pain and lesion growth, showing that blocking this pathway reduces pain and lesion size in mice.

2 citations

𝕏 PostBluesky