Natasha Jordan

This study examines SLE monocyte subsets, finding they are pro-inflammatory and show dysregulated metabolism in response to bacterial stimuli.

This study identifies increased glycolysis in SLE neutrophil subsets, correlating with disease activity and metabolic flexibility.

This study identifies mitochondrial bioenergetic failure in SLE immunocytes, showing reduced OXPHOS across multiple immune subsets and proposes targeting mitochondrial fitness for therapy.