Author

Nicolai S. Henriksen

University of CopenhagenORCID 0009-0008-7647-5535Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Jesper B. BirkiD· 2 shared Jørgen F. P. WojtaszewskiiD· 2 shared Rasmus KjøbstediD· 2 shared Christian K. PehmølleriD· 1 shared David Ernest JamesiD· 1 shared Esben SøndergaardiD· 1 shared Hannah HuckstepiD· 1 shared Henriette PilegaardiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersPhysiology

Jesper B. BirkORCID

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2 papers

Recent·Most cited

PhysiologybioRxivPreprintSep 2, 2026

Personalized phosphoproteomics establish mTORC1 as a regulator of exercise-induced insulin sensitization in human skeletal muscle

Magnus R. Leandersson, Hannah Huckstep, Johan Onslev…·Jørgen F. P. Wojtaszewski

The paper uses personalized phosphoproteomics to show that mTORC1 regulates exercise-induced insulin sensitization in human skeletal muscle through a feedback pathway involving MKNK2 and eIF4G1.

DOI 10.64898/2026.08.27.747624

humanproteomics

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Molecular BiologybioRxivPreprintAug 28, 2023

The human AMPKgamma3 R225W mutation does neither enhance basal AMPKgamma3-associated activity nor glycogen in human or mouse skeletal muscle

Nicolas O. Eskesen, Rasmus Kjøbsted, Jesper B. Birk…·Jørgen F. P. Wojtaszewski

This study investigates the AMPKgamma3 R225W mutation in human and mouse skeletal muscle, finding no increase in basal activity or glycogen levels, but loss of activation by AICAR/muscle contraction.

DOI 10.1101/2023.08.28.555048

humanmouse

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