Author

Hannah E. Vorndran

Cardiovascular Research CenterORCID 0009-0009-0689-4811Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Aidan W. PorteriD· 2 shared Jeffrey L. BrodskyiD· 2 shared Teresa M. BuckiD· 2 shared Thomas R. KleymaniD· 2 shared Allison E. Marciszyn· 1 shared Allison L. Marciszyn· 1 shared Anuradha R. Iyer· 1 shared Arohan R. SubramanyaiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersPhysiology

Aidan W. PorterORCID

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2 papers

Recent·Most cited

PhysiologyAmerican Journal of Physiology-Renal PhysiologyPeer-reviewedOct 21, 2026

An endoplasmic reticulum resident molecular chaperone, GRP170, prevents stress-induced glomerular injury

Aidan W. Porter, Allison E. Marciszyn, Hannah E. Vorndran…·Teresa M. Buck

This study shows that GRP170 protects podocytes from stress-induced glomerular injury by modulating the UPR, as demonstrated in a podocyte-specific knockout mouse model.

DOI 10.1152/ajprenal.90091.2026

mouseexperimental assay

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Cell BiologyJournal of the American Society of NephrologyPeer-reviewedOct 1, 2024

Excess dietary sodium partially restores salt and water homeostasis caused by loss of the endoplasmic reticulum molecular chaperone, GRP170, in the mouse nephron

Aidan W. Porter, Hannah E. Vorndran, Allison L. Marciszyn…·Teresa M. Buck

This study shows that excess dietary sodium partially restores electrolyte imbalance and kidney injury markers in GRP170-deficient mice but not weight or tubule integrity.

1 citation

mouseexperimental assay

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