Author

A. A. Tisdale

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cancer Biology· 2

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

A. A. Alahmari· 2 shared M. E. Feigin· 2 shared S. Venkat· 2 shared A. C. Cornwell· 1 shared A. H. Chaubey· 1 shared C. D. Schwarz· 1 shared E. Cortes Gomez· 1 shared E. J. Paterson· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCancer Biology

A. A. Alahmari

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2 papers

Recent·Most cited

CPSF3 inhibition blocks pancreatic cancer cell proliferation through disruption of core histone processing

A. A. Alahmari, A. H. Chaubey, A. A. Tisdale, C. D. Schwarz, A. C. Cornwell, K. E. Maraszek, E. J. Paterson, M. Kim, S. Venkat, E. Cortes Gomez, J. Wang, K. V. Gurova, M. E. Feigin

This study identifies CPSF3 as a druggable target in pancreatic cancer by showing its inhibition disrupts histone processing and cell proliferation.

bioRxiv · May 9, 2022 · PreprintCancer Biology

Alternative polyadenylation drives oncogenic gene expression in pancreatic ductal adenocarcinoma

S. Venkat, A. A. Tisdale, J. R. Schwarz, A. A. Alahmari, H. C. Maurer, K. P. Olive, K. H. Eng, M. E. Feigin

This study identifies APA-driven gene expression changes in pancreatic ductal adenocarcinoma, revealing miRNA loss and therapeutic targets like CK1.

bioRxiv · Aug 30, 2019Cancer Biology