Author

M. E. Feigin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:differential expression· 3 Research Focus:gene regulatory network inference· 3 Organism:human· 3 Approach:statistical model· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

S. Venkat· 3 shared A. A. Alahmari· 2 shared A. A. Tisdale· 2 shared A. C. Cornwell· 1 shared A. H. Chaubey· 1 shared C. D. Schwarz· 1 shared E. Cortes Gomez· 1 shared E. J. Paterson· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersCancer Biology

S. Venkat

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

CPSF3 inhibition blocks pancreatic cancer cell proliferation through disruption of core histone processing

A. A. Alahmari, A. H. Chaubey, A. A. Tisdale, C. D. Schwarz, A. C. Cornwell, K. E. Maraszek, E. J. Paterson, M. Kim, S. Venkat, E. Cortes Gomez, J. Wang, K. V. Gurova, M. E. Feigin

This study identifies CPSF3 as a druggable target in pancreatic cancer by showing its inhibition disrupts histone processing and cell proliferation.

bioRxiv · May 9, 2022 · PreprintCancer Biology

Alternative polyadenylation characterizes epithelial and fibroblast phenotypic heterogeneity in pancreatic ductal adenocarcinoma

S. Venkat, M. E. Feigin

The paper identifies 3 UTR-APA patterns in PDAC single-cell data, linking them to metastatic epithelial and inflammatory fibroblast subpopulations and phenotypic heterogeneity.

bioRxiv · Sep 18, 2021Cancer Biology

Alternative polyadenylation drives oncogenic gene expression in pancreatic ductal adenocarcinoma

S. Venkat, A. A. Tisdale, J. R. Schwarz, A. A. Alahmari, H. C. Maurer, K. P. Olive, K. H. Eng, M. E. Feigin

This study identifies APA-driven gene expression changes in pancreatic ductal adenocarcinoma, revealing miRNA loss and therapeutic targets like CK1.

bioRxiv · Aug 30, 2019Cancer Biology