Author

A. Bykov

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:clustering· 1 Approach:deep learning· 1

Frequent Co-Authors

F. Halbritter· 2 shared A. Dupanovic· 1 shared A. R. Poetsch· 1 shared A. Sdobnov· 1 shared B. Kovacic· 1 shared B. Radic Sarikas· 1 shared B. Sax· 1 shared D. Emminger· 1 shared

Activity by Year

2023-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCancer Biology

F. Halbritter

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

YAP1 is a key regulator of EWS::FLI1-dependent malignant transformation upon IGF-1 mediated reprogramming of bone mesenchymal stem cells

H. Kovar, R. Noorizadeh, B. Sax, T. Javaheri, B. Radic Sarikas, V. Fock, M. Kauer, A. Bykov, V. Suresh, M. Schlederer, L. Kenner, G. Weber, W. Mikulits, F. Halbritter, R. Moriggl

The paper shows that IGF-1 reprograms bone mesenchymal stem cells in EWS::FLI1-mutant mice, leading to tumor formation, with Yap1 playing a central role, and suggests combined IGF-1R and YAP/TEAD inhibition as a potential treatment.

bioRxiv · Jul 15, 2024Cancer Biology

Beyond Life: Exploring Hemodynamic Patterns in Postmortem Mice Brains

A. Sdobnov, G. Piavchenko, V. Tsytsarev, A. Bykov, I. Meglinski

The paper uses LSCI and analytical methods to study cerebral blood flow dynamics in postmortem mice brains, identifying hemodynamic activity localization and temporal changes.

Journal of Biophotonics · May 7, 2024Peer-reviewedBioengineering

Natural killer cell cytotoxicity shapes the clonal evolution of B cell leukaemia

M. C. Buri, M. R. Shoeb, A. Bykov, P. Repiscak, H. Baik, A. Dupanovic, F. O. David, B. Kovacic, F. Hall-Glenn, S. Dopa, J. Urbanus, L. Sippl, S. Stofner, D. Emminger, J. Cosgrove, , E. M. Putz

This study shows NK cells influence all phases of cancer immunoediting in B-ALL, promoting resistant clones through selective pressure and identifying LY6E as a marker of poor prognosis.

bioRxiv · Nov 16, 2023Cancer Biology