Author

A. Ferrantella

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:mouse· 1 Research Focus:spatial domain detection· 1

Frequent Co-Authors

A. Saluja· 1 shared B. C. Durden· 1 shared B. Giri· 1 shared K. Kesh· 1 shared N. S. Sharma· 1 shared R. Hadad· 1 shared S. Banerjee· 1 shared V. Dudeja· 1 shared

Activity by Year

2019-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

A. Saluja

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1 paper

Recent·Most cited

Targeting tumor intrinsic metabolic node in pancreatic cancer causes tumor regression, remodels extracellular matrix and sensitizes to anti-PD1 therapy.

N. S. Sharma, V. K. Gupta, V. T. Garrido, R. Hadad, B. C. Durden, K. Kesh, B. Giri, A. Ferrantella, V. Dudeja, A. Saluja, S. Banerjee

This study shows that targeting the HBP metabolic node in pancreatic cancer with DON reduces tumor growth, remodels the ECM, and enhances response to anti-PD1 therapy.

bioRxiv · Sep 18, 2019Cancer Biology