Author

B. C. Durden

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1

Frequent Co-Authors

A. Saluja· 2 shared K. Kesh· 2 shared N. S. Sharma· 2 shared R. Hadad· 2 shared S. Banerjee· 2 shared V. Dudeja· 2 shared V. K. Gupta· 2 shared A. Ferrantella· 1 shared

Activity by Year

2019-2019 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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2 shared papersCancer Biology

A. Saluja

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2 papers

Recent·Most cited

Targeting tumor intrinsic metabolic node in pancreatic cancer causes tumor regression, remodels extracellular matrix and sensitizes to anti-PD1 therapy.

N. S. Sharma, V. K. Gupta, V. T. Garrido, R. Hadad, B. C. Durden, K. Kesh, B. Giri, A. Ferrantella, V. Dudeja, A. Saluja, S. Banerjee

This study shows that targeting the HBP metabolic node in pancreatic cancer with DON reduces tumor growth, remodels the ECM, and enhances response to anti-PD1 therapy.

bioRxiv · Sep 18, 2019Cancer Biology

Long non-coding RNA GAS5 acts as proliferation brakes in CD133+ cells responsible for tumor recurrence

N. S. Sharma, P. Gnamlin, B. C. Durden, V. K. Gupta, K. Kesh, V. Garrido, R. Hadad, V. Dudeja, A. Saluja, S. Banerjee

This study shows GAS5 regulates quiescence and growth arrest in CD133+ cells, which drive tumor recurrence in pancreatic cancer by inhibiting cell cycle progression and altering metabolic pathways.

bioRxiv · Jun 13, 2019Cancer Biology