Author

A. Gonzalez-Guerra

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Organism:C. elegans· 1 Research Focus:cell signaling & trafficking· 1 Assay:electrophysiology· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1

Frequent Co-Authors

A. I. Moreno-Manuel· 1 shared A. Macias· 1 shared A. Mendana· 1 shared D. C. Volke· 1 shared D. Mazel· 1 shared F. de la Cruz· 1 shared F. J. Bermudez-Jimenez· 1 shared F. M. Cruz· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

A. I. Moreno-Manuel

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Long-term evolution reveals the role of the circadian cycle in the environmental adaptation of cyanobacteria

A. Mendana, M. Santos-Merino, R. Gutierrez-Lanza, M. Dominguez-Quintero, J. M. Medina, A. Gonzalez-Guerra, V. Campa, M. Ducos-Galand, R. Lopez-Igual, D. C. Volke, M. Gugger, P. I. Nikel, D. Mazel, F. de la Cruz, R. Fernandez-Lopez

The paper uses experimental evolution to show that circadian clock mutations in cyanobacteria enhance growth under continuous light by altering transcriptomic and metabolic responses.

bioRxiv · Mar 12, 2024 · PreprintMicrobiology

Dual Dysfunction of Kir2.1 Underlies Conduction and Excitation-Contraction Coupling Defects Promoting Arrhythmias in a Mouse Model of Andersen-Tawil Syndrome Type 1

A. Macias, A. Gonzalez-Guerra, A. I. Moreno-Manuel, F. M. Cruz, N. Garcia-Quintans, L. K. Gutierrez, M. Roche-Molina, F. J. Bermudez-Jimenez, V. Andres, M. L. Vera-Pedrosa, I. Martinez-Carrascoso, J. A. Bernal, J. Jalife

This study identifies a dual dysfunction of Kir2.1 in a mouse model of ATS1, linking it to conduction and excitation-contraction coupling defects that promote arrhythmias.

bioRxiv · Jun 17, 2021 · PreprintCell Biology