Author

A. Hilderink

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

C. G. Burd· 2 shared J. C. M. Holthuis· 2 shared T. Sokoya· 2 shared Y. Kim· 2 shared A. S. Klymchenko· 1 shared B. Sarkar· 1 shared C. J. Clarke· 1 shared D. I. Danylchuk· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersCell Biology

C. G. Burd

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2 papers

Recent·Most cited

Pathogenic variants of sphingomyelin synthase SMS2 disrupt lipid landscapes in the secretory pathway

T. Sokoya, J. Parolek, M. M. Foged, D. I. Danylchuk, M. Bozan, B. Sarkar, A. Hilderink, M. Philippi, L. D. Botto, P. A. Terhal, O. Maekitie, J. Piehler, Y. Kim, C. G. Burd, A. S. Klymchenko, , J. C. M. Holthuis

The paper shows pathogenic SMS2 variants disrupt lipid landscapes in the secretory pathway by trapping sphingomyelin in the ER and altering cholesterol and glycerophospholipid distributions.

bioRxiv · Apr 2, 2022Cell Biology

Ca2+-activated sphingomyelin scrambling and turnover mediate ESCRT-independent lysosomal repair

P. Niekamp, T. Sokoya, L. Vittadello, Y. Deng, Y. Kim, A. Hilderink, M. Imlau, C. J. Clarke, C. G. Burd, J. C. M. Holthuis

The paper identifies a Ca2+-activated sphingomyelin scrambling pathway that mediates ESCRT-independent lysosomal repair by converting sphingomyelin into repair intermediates.

bioRxiv · Mar 12, 2021Cell Biology