Author

A. J. Jang

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

B.-Y. Kang· 2 shared C. Park· 2 shared M. J. Passineau· 2 shared R. L. Benza· 2 shared W. A. Lam· 2 shared C. M. Hart· 1 shared D. H. Lee· 1 shared H. Karmouty-Quintana· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersPhysiology

B.-Y. Kang

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

USP11 promotes endothelial apoptosis-resistance in pulmonary hypertension by deubiquitinating HINT3

A. J. Jang, V. Tseng, J. S. Kim, R. S. Stearman, Y. Zhao, J. Zhao, J. Choi, J. Lister, M. J. Passineau, W. A. Lam, C. Park, R. L. Benza, B.-Y. Kang

This study identifies USP11 as a deubiquitinase that stabilizes HINT3, promoting endothelial apoptosis-resistance in pulmonary hypertension.

bioRxiv · May 17, 2022 · PreprintPhysiology

PPARγ/ETV2 Axis Regulates Endothelial-to-Mesenchymal Transition in Pulmonary Hypertension

D. H. Lee, M. Kim, S. S. Chang, A. J. Jang, J. Kim, J. Ma, M. J. Passineau, R. L. Benza, H. Karmouty-Quintana, W. A. Lam, R. L. Sutliff, C. M. Hart, C. Park, B.-Y. Kang

This study identifies the PPARγ/ETV2 axis as a novel pathway that suppresses endothelial-to-mesenchymal transition in pulmonary hypertension.

bioRxiv · May 17, 2022Physiology