Author

S. S. Chang

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. J. Jang· 1 shared B.-Y. Kang· 1 shared C. Hino· 1 shared C. M. Hart· 1 shared C. Park· 1 shared D. H. Lee· 1 shared G. Chan· 1 shared G. Jordaan· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPhysiology

A. J. Jang

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cellular protection from H2O2 toxicity by Fv-Hsp70 and mutants of Fv-Hsp70. Protection via catalase and gamma-glutamyl cysteine synthase.

C. Hino, G. Chan, G. Jordaan, S. S. Chang, J. Saunders, M. T. Bashir, J. E. Hansen, J. Gera, R. H. Weisbart, R. N. Nishimura

This study shows Fv-Hsp70 and its mutants protect SH-SY5Y cells from H2O2 toxicity through a protein-binding domain, and this protection involves catalase and gamma-glutamylcysteine synthase.

bioRxiv · Feb 22, 2023 · PreprintCell Biology

PPARγ/ETV2 Axis Regulates Endothelial-to-Mesenchymal Transition in Pulmonary Hypertension

D. H. Lee, M. Kim, S. S. Chang, A. J. Jang, J. Kim, J. Ma, M. J. Passineau, R. L. Benza, H. Karmouty-Quintana, W. A. Lam, R. L. Sutliff, C. M. Hart, C. Park, B.-Y. Kang

This study identifies the PPARγ/ETV2 axis as a novel pathway that suppresses endothelial-to-mesenchymal transition in pulmonary hypertension.

bioRxiv · May 17, 2022Physiology