Author

A. M. Manuel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biochemistry· 1 Microbiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:bacteria (other)· 1 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

A. Ambrus· 1 shared A. Cascon· 1 shared A. D. Gloss· 1 shared A. Deutschbauer· 1 shared A. hawks· 1 shared A. Muszynski· 1 shared D. Belnap· 1 shared E. Bleak· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperBiochemistry

A. Ambrus

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

A weaponized phage suppresses competitors in historical and modern metapopulations of pathogenic bacteria

N. Backman, S. M. Latorre, E. Symeonidi, A. Muszynski, E. Bleak, L. Eads, P. Martinez-Koury, S. Som, A. hawks, A. D. Gloss, D. Belnap, A. M. Manuel, A. Deutschbauer, J. Bergelson, P. Azadi, , T. Karasov

The paper identifies a tailocin, a phage-derived element, used by Pseudomonas spp. to suppress competitors in metapopulations, showing its persistence over 170 years.

bioRxiv · Apr 17, 2023 · PreprintMicrobiology

Defective Function of α-Ketoglutarate Dehydrogenase Exacerbates Mitochondrial ATP Deficits during Complex I Deficiency

G. G. Piroli, A. M. Manuel, R. S. McCain, H. H. Smith, O. Ozohanics, S. Mellid, W. E. Cotham, M. D. Walla, A. Cascon, A. Ambrus, N. Frizzell

This study shows that defective α-ketoglutarate dehydrogenase function worsens mitochondrial ATP deficits in Complex I deficiency by impairing TCA cycle components.

bioRxiv · Jan 9, 2020Biochemistry