Author

A. Marino

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Evolutionary Biology· 1 Microbiology· 1 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

C. Beauloye· 2 shared A. C. Calabuig· 1 shared A. Cuomo· 1 shared A. Di Lorenzo· 1 shared A. Geiser· 1 shared A. Ginion· 1 shared A. Haudry· 1 shared A. Mirabile· 1 shared

Activity by Year

2019-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 7 mutual co-author links

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2 shared papersPhysiology

C. Beauloye

Mutual co-authors in cluster

Author profile →

6 papers

Recent·Most cited

When the liver is in poor condition, so is the heart - cardiac remodelling in MASH mouse models

S. Bott, J. Lallement, A. Marino, E. P. Daskalopoulos, C. Beauloye, H. Esfahani, C. Dessy, I. Leclercq

This study examines cardiac remodelling in MASH mouse models, finding adverse structural changes in the heart linked to liver disease, not overfeeding or aging.

bioRxiv · May 1, 2024Physiology

Effective population size does not explain long-term variation in genome size and transposable element content in animals

A. Marino, G. Debaecker, A.-S. Fiston-Lavier, A. Haudry, B. Nabholz

The paper examines the relationship between effective population size and genome size variation in animals, finding no evidence that genetic drift drives TE accumulation across species.

bioRxiv · Feb 26, 2024Evolutionary Biology

Sodium-myo-inositol cotransporter-1, SMIT1, promotes cardiac hypertrophy and fibrosis induced by pressure overload in mice

A. Marino, J. Cumps, L. Guilbert, A. Geiser, C. Baufays, A. Ginion, L. Ferte, S. Battault, F. De Matos, J. Ambroise, C. J. Zuurbier, F. Lezoualc'h, C. Pestiaux, G. Pyka, G. Kerckhofs, , C. Beauloye

This study shows that SMIT1 promotes cardiac hypertrophy and fibrosis in mice under pressure overload by enhancing IP3/Ca2+-dependent signaling pathways.

bioRxiv · Oct 25, 2023Cell Biology

The assembly of cancer-specific ribosomes by the lncRNA LISRR suppresses melanoma anti-tumour immunity

S. Cinque, Y. Verheyden, S. Adnane, A. Marino, V. Katopodi, E. Demesmaeker, Z. Knezevic, S. Hanache, R. Vendramin, A. Cuomo, J. Pozniak, A. C. Calabuig, S. Tabruyn, O. Bechter, M. F. Baietti, , E. Leucci

This study identifies LISRR, a cancer-specific lncRNA that assembles immunosuppressive ribosomes in melanoma, suppressing anti-tumour immunity and resistance to immune checkpoint blockade.

bioRxiv · Jan 6, 2023 · PreprintCancer Biology

Acinetobacter baumannii and cefiderocol between cidality and adaptability

S. Stracquadanio, C. Bonomo, A. Marino, D. Bongiorno, G. F. Privitera, D. A. Bivona, A. Mirabile, P. G. Bonacci, S. Stefani

This study investigates cefiderocol interactions with A. baumannii strains, revealing resistance mechanisms and the potential of beta-lactamase inhibitors to restore drug efficacy.

bioRxiv · May 5, 2022Microbiology

Pressure overload leads to coronary plaque formation, progression and myocardial events in ApoE-/- mice

A. Marino, Y. Zhang, L. Rubinelli, J. E. Ip, A. Di Lorenzo

This study shows that pressure overload in ApoE-/- mice leads to coronary plaque formation, progression, and myocardial events, mimicking human coronary artery disease.

bioRxiv · Feb 26, 2019Pathology