Author

A. Mauri-Crouzet

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Pathology· 1

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Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

B. Ropert· 1 shared C. Cochaud· 1 shared E. C. GENIN· 1 shared F. Lespinasse· 1 shared G. Auge· 1 shared K. Fragaki· 1 shared S. Bannwarth· 1 shared S. Lacas-Gervais· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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1 shared paperPathology

B. Ropert

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1 paper

Recent·Most cited

SLP2/prohibitins aggregates and instability of the PHB complex are key elements in CHCHD10S59L-related disease

E. C. GENIN, S. Bannwarth, B. Ropert, A. Mauri-Crouzet, F. Lespinasse, G. Auge, K. Fragaki, C. Cochaud, S. Lacas-Gervais, V. Paquis-Flucklinger

The paper identifies SLP2/prohibitin aggregates and PHB complex instability as key factors in CHCHD10S59L-related disease, leading to mitochondrial dysfunction and apoptosis.

bioRxiv · May 31, 2021 · PreprintPathology